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Ketamine users made more antisaccade errors than both control groups but did not differ from patients.
Although the main effect condition suggests that across modalities more antisaccade errors were made than prosaccade errors, the interaction type*modality was not significant.
Children with ADHD combined type made more antisaccade errors than control children, while no group differences were found between children with ADHD inattentive type and control children [ 12].
Although both schizophrenia and ASD samples made more antisaccade errors than controls, ASD participants performed correct trials faster and schizophrenia patients performed more slowly.
Given that children with ADHD have difficulties with response inhibition and make more antisaccade errors than children without ADHD, one might assume that activity of frontal structures involved in the generation of antisaccades is altered.
Compared with matched groups of nonketamine polydrug users and non-drug-using control subjects, ketamine users made significantly more antisaccade errors but were no different on spatial accuracy and latency of antisaccades or on accuracy of smooth pursuit.
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This is due to more prosaccade and fewer antisaccade errors in the acoustic than the visual condition.
ASD participants were slightly more likely to correct antisaccade errors [trend: t(22) = 1.77, P = 0.09; HC: 85 ± 13%; ASD: 93 ± 5%] (Fig. 2C), and did not differ from controls in the proportion of long versus short self-corrections [ t(22) = 1.50, P = 0.15, HC: 67 ± 29%; ASD: 51 ± 20%].
Nicotine significantly decreased antisaccade errors (p <.01) in both groups.
Antisaccade errors declined with age, indicating an ongoing development of inhibitory functions.
We classified corrective saccades that followed antisaccade errors into short and long latency self-corrections (c.f.
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