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Six rhesus monkeys were primed intramuscularly with IDLV-Env and boosted with the same vector after 1 year.
Prior to each choice, the monkeys were primed with a brief (500 ms) presentation of an image belonging to one of the four image pools used in the previous experiment (high status male faces, low status male faces, female perinea, or gray square).
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Overall, these data suggest that aglycosyl hu5c8 IC and CDP7657 IC do not induce platelet aggregation in both human and Rhesus monkey test systems; the same was true even when Rhesus monkey platelets were primed with sub-optimal ADP (See Additional file 3).
Lactating rhesus monkeys were intramuscularly primed with either recombinant DNA (n = 4) or modified vaccinia virus Ankara (MVA) poxvirus vector (n = 4) expressing the T/F HIV Env C.1086 and then boosted twice intramuscularly with C.1086 gp120 and the adjuvant MF59.
In this study, five uninfected, hormone-induced lactating, Mamu A*01 female rhesus monkey were systemically primed and boosted with rDNA and the attenuated poxvirus vector, NYVAC, containing the SIVmac239 gag-pol and envelope genes.
To improve vaccine-elicited antibody responses in milk, hormone-induced lactating rhesus monkeys were vaccinated with a transmitted/founder (T/F) HIV Env immunogen in a prime-boost strategy modeled after the moderately protective RV144 HIV vaccine.
The monkeys were screeching.
"And the monkeys were everywhere.
Seventy-three rhesus monkeys were classified into 3 groups: healthy monkeys, monkeys with MetS, and monkeys with T2DM.
Monkeys were its primary host.
Undoubtedly, research on monkeys is now a prime target for campaigners.
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