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To better understand the fate of these receptor molecules, we looked for the enrichment of putative endocytic structures upon blockage of myosin Va.
In addition to these molecules, we looked for expression of cytokines, chemokines, and interleukins as a surrogate marker for the potential of these cells to mount an inflammatory response.
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Since both granzyme and NKG2D are Th1 related immune molecules, we then looked in to the Th1 transcription factor T-bet expression after PKC and NFATc1 inhibition.
In this study, we looked at molecules such as hydroxyl-cinnamic acids (HCAs), which are known to exist as positional and geometrical isomers conjugated to different organic acids namely quinic- and isocitric acid.
In an attempt to understand biochemical pathways regulating EP bridge form and function, we looked at molecules involved in inflammatory airway diseases, such as asthma and chronic obstructive pulmonary disorder.
This equates to 49.4 %, similar to what was found when we looked at all molecules in a patent.
To characterize Treg-cell phenotype, we looked for signature molecules, including CD25, GITR, CTLA-4, and PD-1, which are known to associate with the CD4+ Treg cells.
To explore what might act downstream of PKG in thermotolerance, we looked for potential candidate molecules known to be intermediaries of both PKG and K+ channels.
To gain further insight into the relative contribution of advection and diffusion we looked at the distance molecules will travel via advection for a specific signal strength (S = 1000).
If we look at time per molecule, MOLGEN is 4 times faster than OMG for small molecules like pyruvic acid.
So, we look for complex carbon molecules and we look for water and we look for temperatures at which that water can be a fluid.
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Since I tried Ludwig back in 2017, I have been constantly using it in both editing and translation. Ever since, I suggest it to my translators at ProSciEditing.

Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com