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Despite the existence of multiple alternative costimulatory molecules, the majority of modular designs, used in clinical trials, are based on the CD137 and CD28 signaling domains.
In the presence of these molecules, the majority of hESC-derived EBs differentiated into Msx1+ mesencephalic neural progenitor cells and TH+ neurons after 10 14 days, clearly mimicking the SDIA effect.
Bioinformatics tools were employed to characterize domains and sequence motifs in ten ULBP genes predicted to encode cell surface molecules, the majority of which are predicted glycoproteins.
As the contact line moves over these molecules, the majority (approximately 60%) are stretched, with the remainder detaching from the surface.
Despite the fact that antigen recognition by MH class II is determined by a protein complex including both IIα and IIβ molecules, the majority of studies have focused on only a single component of this complex (typically MH IIβ), neglecting the fact that antigen discrimination is likely to be realized via unique permutations of MH IIα and IIβ alleles in the host.
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Given that those lines are at most 8 10 protein molecules wide, the majority of these isolated points are probably single molecules, their size determined by the point-spread function of the microscope.
Currently, InnateDB only curates interactions involving human and mouse molecules, with the majority of curated interactions (72% or 8,569 interactions) involving human molecules (although there has been no specific focus on human as opposed to mouse).
The free movement of molecules into the majority of brain regions is restricted by the presence of the blood-brain barrier (BBB) and blood-cerebrospinal fluid (CSF) barrier (BCSFB).
Morphogenesis of vasculature relies on processes like cell adhesion, motility, and proliferation that inevitably include the actin cytoskeleton and associated junctional molecules, making the majority of these complexes a requisite of VG, AG, and LAG [ 9– 14].
For one third of the Census proteins (79 in total), structures have been determined in complex with small-molecule ligands; the majority of these proteins are enzymes.
MyD88 is a central adaptor molecule for the majority of TLRs, with the exception of TLR3 (Akira, 2006; O'Neill and Bowie, 2007).
More suggestions(15)
elements the majority
genes the majority
perpetrators the majority
pathways the majority
species the majority
terrorists the majority
medications the majority
sequences the majority
suspects the majority
molecules the synthesis
molecules the reader
molecules the building
molecules the situation
molecules the cleavage
molecules the liner
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