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During the thermal decomposition process, molecule-size pores generated when the HF molecules released from the flower-like particles.
It is thus conceivable that molecules released from humic superstructures may then access cell membranes and induce different physiological responses.
While the microgels are nontoxic, the drug molecules released from the microgels remain active to kill the cancer cells.
So, the triggered catecholamine molecules released from PC12 cells can be continuously monitored through the changes in current flowed on the surface of nanotubes [81].
The HCl addition resulted in transformation of the aniline molecules released from the particles to anilinium cations, which in turn were polymerized by chemical oxidation with APS.
Toll-like receptors (TLRs), such as TLR4, are required for activation of proinflammatory cellular signaling pathways in response to microbial products, but can also recognize endogenous molecules released from damaged tissues.
However, after phosphorylated species (e.g., DNA molecules) replaced RFMP from the surface of the RFMP-Fe3O4@Al2O3 RFMP-Fe3O4@Al2O3wave heating for 15 s, the RFMNPsolecunderreleased fromicrowaves enheatingthe fororescence intensity in the solution.
Recent studies indicate that endogenous molecules released from stressed or dead cells such as heat shock proteins (HSP; TLR2 and TLR4) and high mobility group box 1 (HMGB1; TLR2 and TLR4) are also important TLR agonists (Asea et al., 2002; Kepp et al., 2011).
We found that the electrical charge on the non-adsorbed lipid droplets became less positive or even became negative in the presence of the hydrogel, and that extensive droplet aggregation occurred, which was attributed to the ability of gellan molecules released from the hydrogels to interact with the lipid droplets.
The organic molecules released from the dead bacterial cells stimulate fresh bacterial and algal growth.
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In the danger model [ 1, 2], the self-molecules released from damaged cells (the damage-associated molecular patterns (DAMP)) are sensed by DAMP receptors on immune cells, triggering an inflammatory response.
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