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The interaction between canine platelets or molecules released during platelet activation and these tumour cell lines inhibits their migration, which suggests that canine platelets might antagonize metastasis of canine osteosarcoma.
Pattern recognition receptors (PRRs) are activated by bacterial products and by damage-associated molecular patterns (DAMPs), which are endogenous molecules released during tissue injury [12].
Similar to the release of secretory products during exocytosis, the oxidation current exhibits a spike-like time course with a noninstantaneous rising phase (time between onset of current and maximum flux, t(max)) with approximately 25% of the molecules released during this period.
In some conditions molecules released during the apoptotic response can activate the NF-κB pathway.
Endogenous molecules released during tissue injury can trigger an innate immune response and are termed damage-associated molecular patterns (DAMPs).
The biological properties of the bECM material suggest that the constituent molecules released during in vitro scaffold degradation enhance cell proliferation.
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The extra peak may also be an unidentified molecule released during mechanical perturbation.
TREM-1 may be involved in the amplification of the inflammatory response in AP, and its ligand could be an endogenous molecule released during cellular damage associated with AP [ 24].
Although at this stage the fat body would have had direct contact with parasites or at least molecules released by parasites during midgut egress, the transcriptional response in the fat body was more pronounced at day 1 than on day 11 post infection.
Fetal exposure to allergens or molecules released by various infections during pregnancy has been an area of intense study over the past several decades.
The molecules released into the circulation occur during brain ischemia [ 21], indicating disassembly of TJs, and then disrupt the integrity of the BBB.
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