Note that the 3-D reconstruction offers more information, including the depth and size of the tumor, and the possibility to determine the exact number of fluorophore indicator molecules (or released drug), allowing for quantitative molecular imaging.
In contrast, paclitaxel by itself does not enhance transcription of IL-1β and TNF-α mRNAS or increase translation of the pro-IL-1β precursor molecule, or release of IL-1β and TNF-α (Allen et al, 1993).
Vascular PMN accumulation showed no spatial correlation with increased vessel permeability, enhanced expression of endothelial cell adhesion molecules, platelet aggregation or release of neutrophil extracellular traps.
The SF FSTL1 levels were increased significantly after treatment or following several cycles of freezing and thawing (data not shown), suggesting that FSTL1 could be tethered in a large molecule complex or released from viscous contaminants.
Pathogens synthesize proteins that can serve as "recognizable" antigens; they may express the molecules on their surface or release them into the surroundings (body fluids).
RAN interacts with importin [VectorBase: AAEL007521] and exportin to change their ability to bind or release cargo molecules for the transport through the nuclear pore.
Damage-associated molecular patterns (DAMPs) are molecules released or exposed by dead, dying, injured, or stressed non-apoptotic cells, with multiple roles in inflammation and immunity.
15, 39, 43 Parasites can stimulate intracellular signaling events in endothelial cells whether through direct adhesion to receptors such as CD36 or intercellular adhesion molecule 1 (ICAM-1) 45, 45 or release of soluble factors.
These carbon monoxide-releasing molecules, or CO-RMs, have diverse biological effects, similar in many respects, but not identical, to those of CO [9], including vasodilation [10 12], reducing inflammation [13,14] and cardiac graft rejection [15,16].
Biomarkers are molecules or fragments that are released into biologic fluids during the process of tissue turnover and, for rheumatoid arthritis (RA), are considered to be indicative of degradation or synthesis of cartilage, bone, and synovial tissue [ 1].
