Sentence examples for molecules described above from inspiring English sources

Collectively, our results suggest that a high degree of hydration of these small molecules (and their counterions where appropriate) is a prerequisite for a potent inhibitor of ice recrystallization.?A number of these small molecules described above have been investigated as cryoprotectants and have been found to freeze human red blood cells (RBCs) using low amounts of glycerol.

While the two small molecules described above are pathway inhibitors, small-molecule epigenetic modulators are also receiving considerable attention as a possible therapeutic option.

In addition to the signaling molecules described above, various molecules secreted by adipose tissue influence the relative allocation of energy between these ends.

ECM components, matrix-degrading proteases, and cellular adhesion molecules described above may be involved in EC emigration, sprouting, and thus angiogenesis.

While the tumor size is small, macrophages from the surrounding tissue accumulate in and around the tumor by tumor cell-derived chemotactic molecules described above, and TAMs derived from the surrounding tissue macrophages account for the majority of TAMs.

Our increasing knowledge of the mechanisms determining sensitivity to Top 1 inhibitors raises the possibility that some of the key molecules described above will have utility as biomarkers that predict response of tumours to treatment.

To test this hypothesis, we took the well-known OKT3 anti-CD3 variable domains and combined them with domains derived from Cetuximab (anti-EGFR) to create three different formats of DVD-Ig molecules, i.e., huIgG1-wild-type, huIgG1-mutant (L234A, L235A), and the anti-EGFR/anti-CD3 Half DVD-Ig molecule described above.

The intensity-based small-molecule probes described above have been instrumental in revealing exciting new processes in Zn2+ biology.

The experimental design described above was repeated.

Any of the molecules described in the discussion above could represent a potential therapeutic target in the treatment of SSc, with therapies targeting each of the three cardinal features of CTD: inflammation/autoimmune activation, vasculopathy and fibrosis.

HUVECs were transfected with pre-miR or LNA molecules as described above.