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These latter studies suggest that small molecules could potentially be used to disrupt CDI toxin/immunity complexes.
Any unstable system of potentially reactive molecules could potentially serve as a way of storing free energy, if the cell maintained their concentration far from the equilibrium point of the reaction.
Blocking of either of these molecules could potentially lead to undesired side effects on bone regeneration.
Other proteins and small molecules could potentially account for the observed superoxide detoxification activity (Figs. 4 and 5).
This indicates that T cells from individuals bearing such HLA molecules could potentially develop broad immune responses to vaccination with the conserved epitopes of HIVBr18.
They suggest that, for the MFS efflux systems, all molecules could potentially inhibit the active site; however, 5'-OCQA exhibits in both cases a substantially smaller interaction region showing less complementarity.
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The molecule could potentially treat neuropathic pain, in which even the slightest touch can hurt (pages 1383 1389).
This molecule could potentially be useful in monitoring and in providing clues in aetiology and pathophysiology, and may have implications for the clinical complications associated with PIH.
A significant negative correlation between a molecule-induced signature and disease-induced signature indicates that this molecule could potentially reverse the disease signature.
Principally in both assays (binding to Her2-positive cells or to Her2-coated tips), the homodimeric Fcab molecule could potentially interact with two Her2 molecules.
The latter interaction could be thermodynamically favorable since the polymer molecule could potentially provide several coordinating bonds for a Zn2+ ion (which can form up to 6 of them [ 128]).
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