Sentence examples for molecules are probably from inspiring English sources

One is that these molecules are probably the precursors of life.

Other molecules are probably needed for it to bind β-glucan.

Third, although EFK16-II is capable of stabilizing neutral ellipticine molecules, the amount of stabilized molecules are probably low; the release rate can be slow due to a possibly stronger hydrophobic interaction between neutral ellipticine and EFK16-II in the complexes.

Since multiple types of GR molecules are probably co-expressed in Gr66a-Gal4 expressing labellar taste neurons [25], [27], and the GR66A receptor molecule is involved in the detection of caffeine [26], this suggests that 7-T and perhaps other bitter substances are detected by other, unknown receptor molecule(s).

These molecules are probably regulated in response to immediate biological circumstances and are 'switched' on or off depending on the immediate need of the mosquito.

More likely, the amount of cofactor molecules available at rest is to some extent limiting, as most molecules are probably buffered by the vesicle cluster.

While a complete map of the interactions among the tight junction molecules is probably far from being complete, the available evidence already allows outlining the general molecular architecture of the tight junctions.

We can also find that there is more than one molecule in the asymmetric unit and we will obtain a SMILES with repeated molecules if we treat the list of atom "as is": a SMILES with repeated molecules is probably harmless for many applications (for example, for substructure searches), but is not the best representation of the compound.

As pfcrt is also a putative drug/metabolite transporter, changes in expression of various transporting molecules was probably not random, suggesting that mutations in PfCRT might disturb parasite transporting activities; and the parasites responded by altering expression of various transporters, particularly those transporting H+ and lipids.

Thus, the relative impact of conventional CRRT on inflammatory molecules is probably small and the impact on clinical outcomes uncertain.

Independent of any model, direct experimental proof such as size-dependent blocking with probe molecules is probably most suited to determine the pore dimensions.