Yet, much of the literature examining the molecular response to stroke has focused on targeted pathways that have been well-characterized.
Sugars generated in the shoots and transported through the phloem are involved in establishing a physiological, biochemical, and molecular response to −P stress in plants (Hammond and White 2008).
The molecular response to IFNβ differed significantly between patients in the pattern and number of regulated genes.
We hypothesize that targeting and correcting genes involved in the disparate molecular response to hypoxia may be a novel therapeutic strategy in DMD.
We hypothesize that the molecular response to type I IFN identifies a pathogenetically distinct subset of MS patients whose disease is driven in part by innate immunity.
These results suggested that treatment response status could not be attributed to attenuation of the molecular response to IFNβ over time.
The findings can be extrapolated to IFNβ treatment generally because the molecular response to the three approved IFNβ preparations is highly consistent [26] [30].
Apparently, the molecular response to a single gene mutation is of considerable complexity, and certainly much more complex than detectable using current experimental approaches.
This observation provides the basis for further investigation as to which families of genes may be represented in both the PB molecular response to radiation and chemotherapy.
Taken together, these results suggested that the PB molecular response to ionizing radiation is distinct depending upon which portion of the body is exposed.
