In short, our argument is that it is much more easy to discern topology of form then the underlying developmental and molecular processes that produce form, and therefore, a nomenclature based on topology will be much more stable, and in turn, more useful.
For example, auto-catalytic processes producing monomers make "monomer space" available for molecular evolution.
At the cellular level, it is a molecular process that produces a high-energy molecule called adenosine triphosphate (ATP) using the biochemical energy stored in sugars, fatty acids, and other nutrients.
The two secondary treatment processes produce DOM with distinct molecular compositions, while the secondary clarifiers and UV disinfection unit result in minimal changes in DOM composition.
Different speciation processes produce predictable outcomes in the molecular, morphological, and geographic relationships among lineages.
These observations thus provide critical insight into the molecular processes and mechanisms that produce liver damage and fibrosis in ALD.
When we increase k D and γ, while lowering δ, corresponding to the molecular processes described above, our model produces an oscillatory solution in accordance with the experimental observations (see Figure 2A).
This process produces a formal 'Molecular Disease Model' (MDM) that can be used by clinicians to guide treatment decisions, and refined by researchers based on clinical outcomes and laboratory findings.
Several molecular processes will be discussed below that may be interacting to produce the patterns discussed so far, but we are far from believing to be providing a fully compelling scenario.
It is essential to understand the molecular processes underlying long-term memory to provide therapeutic targets of aberrant memory that produce pathological behaviour in humans.
