Sentence examples for molecular premises from inspiring English sources

In contrast, an account of theory reduction such as the Nagel-Schaffner approach is committed to deduce a description of a higher level phenomenon from purely molecular premises.

The dependence of molecular features on a cellular and organismal context poses problems for theory reduction approaches maintaining that higher level phenomena can be deduced from purely molecular premises.

Proponents of theory reduction such as Nagel (1961) and Schaffner (1976) have replied that a molecular reduction can take the relations of parts and the context of molecular processes into account, e.g., by specifying the relevant context as initial conditions in the molecular premises from which the higher level state is to be deduced (Frost-Arnold 2004).

The underlying reason for phase-separations ('self-ordering') are non-covalent molecular forces (our premise 4), which are also relatively well understood.

In support of this premise, molecular phylogenetic evidence from nuclear and mitochondrial DNA sequencing indicates that crocodilians and birds are indeed each other's closest living relatives [ 5, 6].

Our GO annotation results suggest an enrichment of ATP binding activities under molecular function, supporting the premise that an activation of NBS type motifs occurs in the R genotype during B. glumae challenge.

The author starts with the premise that molecular trees are insufficient for this task; this is especially true for the paralog-rooted trees on which the currently widely-accepted – indeed, assumed – root placement (between eubacteria and archaebacteria) are based.

I stand in another ho-hum field outside Kidlington, the premises of a molecular genetic company off to one side, and ask again the question that keeps coming up in this debate – would it be so terrible to build on it?

The derivation demanded by theory reduction for molecular biology requires that the premises contain a purely molecular specification of the total context.

This premise is consistent with our molecular analysis, which revealed that the NP, PA and PB1 genes have begun to acquire several mammalian host-specific markers (Supplementary Table S3).

Based on the premise of shared genome-wide molecular activity, Iorio et al. [22] systematically compared all the cMap drugs in a pair-wise fashion, rather than searching for drugs eliciting an SDE similar or opposite to an input signature.