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Its molecular pathogenesis is poorly understood.
Its molecular pathogenesis is unclear, and deep sequencing may help identify causative low-frequency variants in tumors.
Their molecular pathogenesis is still largely unknown, and consequently, the best therapeutic management also remains to be determined.
The liver toxicity caused by nano-TiO2 has been reported [8, 12], but its molecular pathogenesis is not known.
Diabetic nephropathy (DN) is a leading cause of end-stage renal disease worldwide, but its molecular pathogenesis is not well defined, and there are no specific treatments.
Despite considerable pathogenic potential of the causative organisms and the clinical relevance of infections with oral streptococci, their molecular pathogenesis is barely understood.
Similar(41)
Although it is known that nano-TiO2 or other nanoparticles can induce serious liver toxicities, the mechanisms and the molecular pathogenesis are still unclear.
Although it is known that nano-TiO2or other nanoparticles can induce liver toxicities, the mechanisms and the molecular pathogenesis are still unclear.
Therefore, new therapeutic agents based on the specific molecular pathogenesis are in great demand.
Important advances in the molecular pathogenesis were obtained through the study of the role of the B-cell receptor (BCR) and the microenvironment.
This probably reflects the complex and multi-factorial nature of GBS virulence and molecular pathogenesis, which is likely to be attributable to multiple genes, polymorphism, and differential expression of these genes and regulation mechanisms [ 24, 25, 29– 329.
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