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Malignant pleural mesothelioma (MPM) is a rare malignant disease, and the understanding of molecular pathogenesis has lagged behind other malignancies.
Understanding this molecular pathogenesis has been a major aim of kidney organogenesis research [1].
Their molecular pathogenesis has not been entirely elucidated.
Until now, its molecular pathogenesis has remained unknown.
In recent years, the rapidly expanding knowledge of cancer molecular pathogenesis has provided new targets for anticancer treatment.
Over the past decade, enhanced understanding of the molecular pathogenesis has led to the development of biologic agents that target specific parts of the immune system.
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Recent insights into molecular pathogenesis have demonstrated that HIV-1 proteins can directly lead to cancer growth by interfering with cellular functions.
Efforts to increase our understanding of the mechanisms of human disease from the perspectives of both gross pathology and molecular pathogenesis have relied heavily on the use of animal models that are assumed to mimic those pathological states.
Although multiple myeloma (MM) remains an incurable disease, there has been a concerted effort toward understanding its molecular pathogenesis, which has paved the way for the development of highly effective, novel therapeutic agents such as the immunomodulatory agents thalidomide and lenalidomide, and the proteasome inhibitor bortezomib.
So far, the molecular analysis of ALS pathogenesis has been hampered by the lack of suitable cell model systems.
Recent advances in genomics and molecular pathogenesis studies have determined that many diseases are caused by a multiplicity of factors.
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