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This work highlights some of the molecular modifications contributing to functional defects of human TIL.
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Genetic modifications contribute to the occurrence of prolapse (proof level 2).
Emerging evidence suggests that aberrant epigenetic modifications contribute significantly to tumor formation and progression.
We propose that these modifications contribute to its antiviral effect.
Whether other post-translational modifications contribute to the regulation of Gli protein activity has been unclear.
Thus, as the important epigenetic modifications, DNA methylation and chromatin modifications contributed to the epigenetic disorders of T2D together.
Simple molecular modifications, especially those aimed at reducing hydrophobicity [56], can often reduce efflux [57, 58].
Diapause evokes a number of physiological, morphological and molecular modifications.
CA is a complex process that involves cellular, physiological, metabolic and molecular modifications.
These molecular modifications promote inhibition of cell migration and potentially restoration of tumour cell contact inhibition.
Our findings show that (a) molecular evolution of paralogs correlates with their expression pattern; (b) gene diversification is obtained through massive genomic rearrangements; and (c) splicing modification contributes to the functional specialization of novel genes.
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