Sentence examples for molecular mechanisms include from inspiring English sources
The underlying molecular mechanisms include interference with viral translational and replication machineries as well as host anti-viral defense.
Molecular mechanisms include large deletions, maternal uniparental disomy for chromosome 15 or mutations involving the imprinting center.
These molecular mechanisms include a sequence for cytoplasmic localization signaling [24], PTEN phosphorylation and involvement of Ras-related nuclear protein (Ran -GTPase activity [25], inteRan -GTPaseh mactivitylt protein (MVP) via a bipartite NLS [25] [22], mono-ubinteractionn [19], S6K-mediated export [26], and passive diffusion [25].
The molecular mechanisms include the transcriptional modulation of adhesive and antiadhesive molecules, proteases and angiogenic factors.
The molecular mechanisms include transcriptional modulation of adhesive and anti adhesive molecules, proteases and angiogenic factors [ 28].
Possible molecular mechanisms include 12-lipoxygenase (12-LOX) [ 19], PI3K/Akt [ 18, 20], MEK/ERK [ 22, 23], and NF- κB [ 24] transduction pathways.
Thus, epigenetics refers to a wide range of molecular mechanisms including DNA methylation of cytosine residues in CpG dinucleotides and post-translational histone modifications.
We find that the transcriptional repressors KNIRPS, KRUPPEL and GIANT are able to restrict reporter gene expression to the posterior abdominal segments, using different molecular mechanisms including short-range repression and competitive binding.
A range of molecular mechanisms including the subunit composition of the receptor(s), phosphorylation and local steroid metabolism, underpin the region- and neuronal selectivity of action of neurosteroids at synaptic and extrasynaptic GABAA receptors.
The study of peptide-membrane interactions is important for understanding a wide range of fundamental molecular mechanisms, including the action of antibiotic peptides, the association of proteins involved in cell signaling, and membrane fusion.
In this review, we analytically summarize recent findings, which indicate that gain-of-function (GOF) mutant p53 proteins counteract the autophagic machinery by various molecular mechanisms including the regulation of AMPK and Akt/mTOR pathways, autophagy-related genes (ATGs), HIF-1α target genes, and the mitochondrial citrate carrier CIC.
