At the molecular level, diapause is less studied but characterized by a complex and regulated change in gene-expression.
Nanotechnology seeks to control matter at the molecular level of scales of less than one micrometer (or one millionth of a meter).
At the proximate (i.e. molecular) level several more or less plausible factors can explain the sex-specific telomere length patterns observed in somatic tissues.
However, for describing interactions at the tissue, cellular, and molecular level, this process is arguably less intuitive, in part because the relationships between these types of interactions and logic gates are less obvious (e.g., it is initially unclear how "binding a receptor" and "intracellular degradation" can be described with logic gates; see Supporting information).
Thus, while our knowledge at the molecular level has vastly increased, much less is known about the macroscopic functioning and organization of the organisms that first evolved nervous systems, or about the ecological circumstances under which they lived.
Although significant inroads have been made in understanding the regulation of secondary growth at the molecular level in dicots (particularly Arabidopsis), far less is understood about these processes in conifers.
The VNAR shares many of the properties of the well-characterised single-domain camelid VHH but is much less understood at the molecular level.
Scientists know a lot about neurons, but less, especially at the molecular level, about astrocytes and their interactions with neurons.
However, spectroscopic analyses of complex mixtures such as resins provide only a broad "fingerprint" and are, therefore, significantly less reliable than detailed molecular level characterization for chemotaxonomic purposes.
The reverse process mesenchymal-to-epithelial transition (MET), much less characterised at the molecular level, restores the specific epithelial identity.
