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To characterize HLR at a molecular level, expression profiles of DR-related genes were examined by quantitative PCR analysis.
In the molecular level, expression of MDM2 was increased and expression of P53 was reduced in QSYQ group, compared with those in the model group.
At the molecular level, expression levels of proteins directly involved in energy metabolism may be increased in highly mobile fish species.
At the molecular level, expression analysis of Pitx1-null and Pitx1+/- mice allowed one to identify and characterize a novel molecular cascade involved in OA pathogenesis.
On a molecular level, expression of GDF-15 is controlled by the ubiquitous transcription factor p53, which responds to stressors such as hypoxia, ischemia, oxidative stress, and inflammation.
At the molecular level expression of the lipid metabolic genes Acaca, Acly and Fasn were lower in liver tissue from HG versus control dams on the first day of lactation.
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At molecular levels, expression of key proteins such as HER2/neu and p53 in breast and non-small cell lung cancer cell lines and their corresponding tumors have been assessed using immunohistochemistry [ 5, 6].
Furthermore, textual information may not reflect the potential specificity and heterogeneity that are concealed in the molecular-level expression measurements of these samples.
At the molecular level, UCP1 expression, together with the expression of nuclear receptors PPARα, PPARγ, and fatty acid transporter aP2 is similar in both knock-out and wild-type animals.
ALL has been studied at the molecular level through expression profiling resulting in un-validated expression correlates of these prognostic indices.
At molecular level, TG2 expression resulted in loss of E-cadherin and increased the expression of various transcriptional repressors (Snail1, Zeb1, Zeb2 and Twist1).
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