It provides an effective way to explain the sensing mechanism at the molecular level by analyzing the adsorption energy, states of density, and energy band structure.
We further aimed to investigate the potential mechanisms underlying the observed circadian behavioral phenotype at the molecular level by analyzing clock gene expression in hippocampal tissue of HAB and NAB mice.
MET was confirmed at the molecular level by analyzing vimentin, snail and twist expression, which are established markers for mesenchymal tumor cells and are upregulated upon epithelial to mesenchymal transition as well as during the metastatic process of epithelial cancers [ 8, 61].
To understand the effects of exposure to DHC on the production of pigment and citrinin at the molecular level, we analyzed the expression level of MpigA and MpksCT encoding polyketide synthases responsible for the synthesis of pigment and citrinin, respectively.
To understand how two subpopulations of these excitatory and inhibitory neurons are regulated at the molecular level, we analyzed specific mRNA expression profiles for receptors that are significantly involved in synaptic transmission and in the synthesis and storage of the principal inhibitory neurotransmitter, γ-aminobutyric acid (GABA).
At the molecular level, we analyzed the expression of microtubule-associated protein1 light chain 3 (LC3) and caspase-3.
Previous studies have already been conducted at the molecular level to analyze the interaction between H. annuus and P. halstedii [ 4- 6, 8, 9].
At the molecular level, previously analyzed species were very distant from each other [ 3], and nucleotide turnover at putatively neutral sites was saturated, preventing the application of several molecular evolution tests.
In an attempt to understand underlying reasons for the varying pathogenicity between HIV-1 and HIV-2 at the molecular level, we analyzed the differences in codon usage pattern of all nine coding genes.
To further characterize the ETP-ALL on the molecular level, we analyzed the expression of selected genes known to be involved in the pathogenesis and with prognostic implications in T-ALL in samples with sufficient material.
