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Electrophysiological, morphological and molecular differences were also identified between these two neuronal types.
To discriminate these two similar species, morphological and molecular differences were analyzed.
Despite the global similarity between iPSCs and hESCs, the profiles were not completely indistinguishable, which led us to study what the molecular differences were.
These molecular differences were also reflected at the cellular level.
Few molecular differences were observed between grade 2 and 3 tumors when using the International Federation of Gynecology and Obstetrics (FIGO) grading system, suggesting their molecular similarity.
Moreover, molecular differences were found between hrHPV-positive and hrHPV-negative HNSCCs, supporting the idea of two separate carcinogenic pathways to HNSCC, one determined by life-style carcinogens and the other by hrHPV [ 5, 6, 10- 13].
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Since we showed in the present study that Tregs, Th17 cells and other IL-17+ cells also show opposed correlations in cervical adenocarcinoma than in squamous cell carcinoma, this suggests that the molecular differences are correlated with a different type of immune response.
Through the comparison of HOC-EC and HA-EC gene expression we were able to demonstrate that tumor-associated EC differ from those in normal tissues, thus proving that molecular differences are maintained in vitro.
Similarly, distinct areal and molecular differences are also evident in developing cortical radial glial cells [47].
Moreover, the observed molecular differences are not direct measures of cognitive differences between sexes.
While syncytia appear similar at the anatomical level at 3d, molecular differences are present [ 20].
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