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Nobody doubts that the physical properties of individual proteins and their ionic environment are what cause molecular complexes to self-assemble and assume their functional shapes.

Examination of the distribution of the various compositional signatures enabled to dissect and monitor the differential sensitivities of the various molecular complexes to perturbation altering mechanical forces exerted by actin (Figs. 4, 5 and Supplementary Figs. S1, S2, S3 and S4).

When these sequences are transcribed and processed into small RNAs, the small RNAs can then guide repressive molecular complexes to invading nucleic acids in trans.

Physically, the "basal" transcription of genes might reflect "nonlinear responses" of the independent gene transcription molecular complexes to internal or external fluctuations including thermal molecular motion.

Similarly, I predict that there will be proteins to stabilize the ribosome to permit the otherwise tenuous interactions within the molecular complexes to continue with high fidelity and efficiency.

The decreased cell motility after LASP-1 silencing can be explained by the functional loss of zyxin as a scaffolding protein that facilitates the formation of molecular complexes to promote site-specific actin assembly required for cell migration.

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With model calculation based on measured polymer extensibilities we were able to identify the optimum combination of cantilever stiffness and anchor length for a given bond strength of the molecular complex to be investigated and to estimate the resulting resolution limit of the different combinations.

Second, in our model, expressions of different genes are assumed to be independent with each other, but it is not the truth since functionally related genes could form a pathway or molecular complex to execute a specific biological process.

The overall better oral absorption of SMV-liposomes as compared to SMV/G5-NH2 molecomplexesplexes appeared to arise from better liposomal solubilization and encapsulation of SMV and more efficient intracellular SMV delivery.

Similar to the GEFs, there are various Rap-GAPs that are specifically targeted to different molecular complexes at various cellular locations [ 59].

As a part of the same machinery, actin-tethered molecular complexes appear to be highly integrated, and they cooperate to regulate organization or disorganization of particular adhesion structure.

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