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Alcohol causes chronic irritation and inflammation of the esophageal mucosa, and consequently induces a series of molecular changes, triggering carcinogenesis.
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We suggest that these molecular changes trigger thymic epithelial senescence accompanied by adipose involution.
To test the basis of CD8+ cell collapse, we compared molecular changes triggered by the virus in splenic CD8+ cells isolated from RAPs and those that did not (regular progressors, REGs), with CD8+ cells from uninfected controls.
It has been proposed that these molecular changes must trigger events that ultimately lead to the clinical phenotype.
This study was designed to identify the molecular basis of phenotypic changes triggered by chronic hypoxia.
The aim of this study was to identify the molecular basis of the phenotypic changes triggered by chronic repeated hypoxia.
We hypothesized that dPAS sensors containing both partners of an ABA co-receptor complex might exhibit larger ABA-dependent ratio changes due to larger conformational changes triggered by ABA-dependent intra-molecular interactions between two ABA binding domains.
The uterine changes triggered by estrogens are directed by numerous genes, but little has been known about the molecular events involved and how they relate to observable physical change.
These huge changes triggered geological mayhem.
These aberrant changes trigger gastric cancer development.
Nerve lesions caused directly by the virus or the inflammatory response can trigger molecular changes in nociceptive neurons leading to channelopathies contributing to the experience of pain.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com