Sentence examples for molecular changes mediated from inspiring English sources

With the exception of BR where some information has been obtained [ 10- 12], molecular changes mediated by ABA, ET, JA and SA that lead to thermotolerance are largely unknown.

Because the mesothelial cells are the primary target cells of asbestos-induced molecular changes mediated through an oxidant-linked mechanism, we used normal mesothelial and malignant mesothelial cells to investigate alterations in molecular signaling in response to a commercially manufactured SWCNT.

Because mesothelial cells are the primary target cells of asbestos-induced molecular changes mediated through an oxidant-linked mechanism, we used well-characterized SWCNTs with a known concentration of metal catalyst contamination to investigate the alterations in molecular signaling in response to SWCNT exposure in normal mesothelial (NM) and malignant mesothelial (MM) cells.

These molecular changes were mediated by iNOS/netrin/PKC signaling pathway downstream of Shh gene overexpression which combined with stem cell transplantation could be a promising strategy for the treatment of an infarcted heart.

Studies are ongoing to investigate the role of molecular changes in mediating the effects of advancing paternal age on social and exploratory behaviors in offspring, by assessing de-novo CNV events and alterations in DNA methylation.

We also performed microarray experiments in PC12 cells to gain insight into the molecular changes that may mediate this phenotype.

In order to gain insight into temperature mediated molecular changes, high throughput de novo transcriptome sequencing and analyses were carried out at 15°C and 25°C, the temperatures known to modulate picrosides content.

Serial (non-terminal) biopsies of tissues such as skeletal muscle could provide corresponding information on the molecular and biochemical changes mediating the effects aging and interventions on function.

T-cell anergy refers to the inactivation of lymphocytes, which undergo intrinsic molecular changes that prevent them from mediating effector functions (Jenkins and Schwartz, 1987; Choi and Schwartz, 2007).

Moreover, since the expression of miRs is highly tissue and disease-specific, changes within the miRNA sequence can indeed specifically predispose to cancers of particular organs and mediate different molecular changes in different tissues (de la Chapelle and Jazdzewski 2011).

In other words, the antimnemonic actions of these manipulations may result from their ability to at least partially reverse the cellular and molecular changes, including synaptic growth, that mediate the expression, rather than the storage, of LTM.