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Molecular analysis confirmed the site-specific insertion of seven transgenes and homoplasmy.
Molecular analysis confirmed the presence, integration, copy number of the transgene all the independent transgenic events.
Consistent with these observations, the molecular analysis confirmed the presence of up to 9 established adhesin genes in the RT-associated S. aureus strains, among them the ones encoding the well-known adhesins FnbA and FnbB involved in eukaryotic cell internalization [40], [52] [54].
Molecular analysis confirmed that the genomic excision was site-specific and conservative.
Molecular analysis confirmed the presence of a mutation in the PTEN tumour-suppressor gene.
Molecular analysis confirmed presence of a metallobetalactamase of GIM-1 genotype.
Similar(44)
Molecular simulation analysis confirmed that the compounds exert their inhibitory activity through binding tightly to hemagglutinin (HA2) protein near the fusion peptide and prevent virus entry.
Molecular genetic analysis confirmed that the prevalence of a key vancomycin resistance gene plummeted from 5.7%to0.8%8%.
Molecular genetic analysis confirmed HNF4A mutation (p.M116I, c.317G>A) as an underlying cause of hyperinsulinaemic hypoglycaemia.
Molecular genetic analysis confirmed IVS13T>G as the most common mutation, but we also identified three novel point mutations.
Molecular epidemiology analysis confirmed that conjugative isolates are not clonal and transfer of antimicrobial resistance was acquired through horizontal transfer of plasmids.
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