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During the last decade, molecular analyses demonstrated that alternative splicing determines the binding properties, intracellular localization, enzymatic activity, protein stability and post-translational modifications of a large number of proteins [9].
Molecular analyses demonstrated the presence of a huge bacterial diversity within the mucilage, with a number of ribotypes significantly higher (approximately double; ANOVA P<0.01) than in surrounding seawater (Figure 2B and 2C).
Previous molecular analyses demonstrated that individuals from the subspecies tricycla are genetically differentiated from the two other subspecies [ 24, 35].
Histological and molecular analyses demonstrated that Emilin1 deficiency is associated with the activation of non-canonical (phosphorylated Erk1 and Erk2) and canonical (phosphorylated Smad2 and Smad3) signaling in aortic valve tissue.
In turn, molecular analyses demonstrated that aberrant overexpression of survivin family proteins [ 15] is required to maintain the Non Responder phenotype, opening fresh opportunities for rational combination regimens to restore anthracycline sensitivity in these patients.
Rigorous assessment of cell fraction purity by both microscopy and molecular analyses demonstrated that the CM fraction was almost completely clear of NM contamination, whereas the NM fraction routinely contained a low level of contaminating CM cells (see Materials and Methods, and supplementary material Fig. S2).
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Molecular analyses demonstrate that both temperature and manure-to-food waste ratio greatly influenced the bacterial communities, while archaeal communities were mainly influenced by temperature.
Some species of Heimioporus have spores with apical pores, but molecular analyses demonstrate that Heimioporus is only distantly related to Spongiforma.
Molecular analyses demonstrate that E74A protein shows diurnal changes in abundance, similar to LARK.
These results were consistent with the molecular analyses demonstrating that nof1-2 is a null allele (see below) with abnormal ovule development (Figure 1c).
Moreover, the molecular analyses demonstrating the effectiveness of antagomir treatment were performed at very short times after antagomir injection, whereas we were able to prove an effective downregulation of miR-221/222, and a consequent upregulation of p27, for longer than three weeks.
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