Sentence examples for modulator of diseases from inspiring English sources

Presentations highlighted studies that suggest that nutrition can be a potential modulator of diseases associated with exposure to environ-mental stressors.

These observations collectively implicate that autophagy is an important modulator of disease pathogenesis.

Because of its central role in guiding fetal development and acting as the gatekeeper of maternal environmental exposure, the placenta responds to and is potentially marked in an epigenetic context by environmental insults which suggests that the placental epigenome might serve not only as a record of in utero exposure but also as a mediator and/or modulator of disease pathogenesis[15], [16].

Recent studies have focused our attention on the possible role of WT SOD1 as a modulator of disease initiation.

Although the foregoing evidence is compelling, initial studies to investigate the role of WT hSOD1 as a modulator of disease produced conflicting outcomes.

Variation in gene transcript levels is thought to be an important modulator of disease risk in humans and SNPs that may mediate this variation are thought to be of great functional significance [ 26].

Although some clinical laboratory urine antigen testing for legionellosis is performed in tertiary care centers in Ottawa and Kingston, the magnitude of this difference may, again, suggest that proximity to the Great Lakes is an important modulator of disease epidemiology.

Serum MBL concentration did not differentiate between CDI cases and AAD controls, but among CDI cases, MBL concentration, but not genotype, was associated with CDI recurrence, indicating that MBL acts as a modulator of disease, rather than a predisposing factor.

Particular strengths of the study include genetic crosses between SBMA mice and mice haploinsufficient for NLK that strongly suggest it is indeed a modulator of disease processes and studies establishing that NLK can phosphorylate the disease protein in SBMA, the androgen receptor (AR), at specific sites.

These findings represent the first direct evidence of a critical role for CCR2-driven inflammatory MO recruitment in DMD pathogenesis and additionally identify CCR2 as an important modulator of disease severity, thus indicating its potential to serve as a new therapeutic target in DMD.

In addition, many papers have shown the in vivo application of these cells as modulators of disease progression both in acute and chronic damaged organs.