Sentence examples for modulation of lymphocyte from inspiring English sources

This effort led to the discovery of BAF312 (1- 4-{1-[(E -4-cyclohexyl-3-trifluoromethylbenzyloxyimino]-ethyl}-2-ethylbenzyl -azetidine-3-carboxylic E -4-cyclohexyl-3-trifluoromethylbenzyloxyimino]-ethyl}-2-ethylbenzyl -azetidine-3-carboxylic E -4-cyclohexyl-3-trifluoromethylbenzyloxyimino]-ethyl}-2-ethylbenzyl -azetidine-3-carboxylic E -4-cyclohexyl-3-trifluoromethylbenzyloxyimino]-ethyl}-2-ethylbenzyl -azetidine-3-carboxylic E -4-cyclohexyl-3-trifluoromethylbenzyloxyimino]-ethyl}-2-ethylbenzyl -azetidine-3-carboxylic E -4-cyclohexyl-3-trifluoromethylbenzyloxyimino]-ethyl}-2-ethylbenzyl -azetidine-3-carboxylic E -4-cyclohexyl-3-trifluoromethylbenzyloxyimino]-ethyl}-2-ethylbenzyl -azetidine-3-carboxylic E -4-cyclohexyl-3-trifluoromethylbenzyloxyimino]-ethyl}-2-ethylbenzyl -azetidine-3-carboxylic E -4-cyclohexyl-3-trifluoromethylbenzyloxyimino]-ethyl}-2-ethylbenzyl -azetidine-3-carboxylic E -4-cyclohexyl-3-trifluoromethylbenzyloxyimino]-ethyl}-2-ethylbenzyl -azetidine-3-carboxylic E -4-cyclohexyl-3-trifluoromethylbenzyloxyimino]-ethyl}-2-ethylbenzyl -azetidine-3-carboxylic

Several mechanisms have been postulated to contribute to the anti-inflammatory effect of probiotics in the gut, including competitive exclusion of pathogens, production of antimicrobial agents and organic acids, enhancement of the epithelial barrier function, increase of mucosal IgA secretion, and modulation of lymphocyte and dendritic cell function [5] [6].

Twelve-week interval appears to be sufficiently long to induce significant shifts in CD4 counts, suggesting that these measurements help monitoring modulation of lymphocyte trafficking and cell redistribution in MS patients, temporarily discontinuing natalizumab therapy.

Thus, the modulation of lymphocyte homeostasis through the functional manipulation of CCL17 harbours opportunities for the design and application of novel therapeutics for the treatment of atherosclerotic vascular disease.

Observed significant shifts in CD4 counts – dramatically increasing from the baseline while on treatment and decreasing back to the baseline level off treatment, then rising in a similar manner on natalizumab reinitiation, suggest that these measurements may aid in monitoring modulation of lymphocyte trafficking and cell redistribution.

Modulation of lymphocyte migration through targeting of SELL and CCR7 is proposed to facilitate HIV-1 immune avoidance (Ramirez et al., 2014; Vassena et al., 2015), and downregulation of NTB-A protects HIV-infected cells from NK cell lysis (Shah et al., 2010).

In view of the reports showing that PCV2 infection leads to modulation of T lymphocyte activity [ 23, 24, 29], the present work sought to characterize the T lymphocyte response in animals following infection with PCV2.

These include modulation of T lymphocyte and neutrophil survival [ 10, 11, 14] and immunosuppression in T and B lymphocytes and dendritic cells [ 12, 13, 16].

The modulation of human lymphocyte proliferative responses was demonstrated with a recombinant outer surface protein A (OspA) vaccine preparation for the prevention of Borrelia burgdorferi infection.

These effects include alteration – usually inactivation – by enzymatic cleavage of several chemokines such as substance P and RANTES, and modulation of T lymphocyte proliferation via interaction with the CD3 signaling pathway and binding of adenosine deaminase (ADA) [2], [3].

Moreover, some studies also suggest that modulation of T- lymphocyte phenotype might be one approach to the treatment of neuropathic pain [ 1].