Sentence examples for modulation of inhibitory from inspiring English sources
It is, however, important to underline that our findings cannot be interpreted as a simple modulation of inhibitory control.
It is likely that these effects result both from direct actions of toluene on processes that regulate the intrinsic activity of VTA DA neurons and indirectly via modulation of inhibitory and excitatory inputs that impinge upon these neurons.
Data suggest that attenuation of pain by VNS occurs via inhibition of signalling through afferent vagus nerve fibres to the trigeminal nucleus caudalis (TNC) [19] and via modulation of inhibitory neurotransmitter release, resulting in decreased glutamate levels in the TNC [20, 21].
In addition, modulation of inhibitory receptors on tumor cells interacting with host T cells could be affected.
Furthermore, an impairment of the modulation of inhibitory intracortical neurotransmission as a function of learning has been observed in these patients.
Additionally, a persistent impairment of the modulation of inhibitory intracortical neurotransmission as a function of learning has been observed in these patients.
Since the anatomical evidence indicate the presence of CB1 in immunonegative vGluT1 as well as in Glu-CB1-KO synaptic terminals showing mostly ultrastructural features of inhibitory-like synapses in the alBNST, we next examined the role of CB1 in the modulation of the inhibitory synaptic transmission.
This specificity of our effects argues against a modulation of general inhibitory control but instead suggests a specific alteration of the goal-directed control of behavior.
These pharmacological manipulations show that modulation of disynaptic inhibitory loops by cholinergic inputs is mediated almost exclusively by heteromeric nAChRs and not by mAChRs.
The contribution of the presynaptic CB1-mediated modulation of the inhibitory synaptic transmission to the functional role of the anterolateral BNST remains to be elucidated.
This suggests that IGF-1 action in mediating EE effects could be exerted through the modulation of intracortical inhibitory circuitry and PNN development.
