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This and the reduction in vascular tissue effects observed with BRL 37344 suggest that uterine β3-adrenoreceptor modulation may provide a novel scientific approach to tocolysis with fewer vascular adverse effects.
They also suggest that neuro modulation may provide an efficient means of targeting regions in parameter space with significant effects on response properties.
These abnormal parameters emerge as therapeutic targets, and their modulation may provide beneficial effects on future cardiovascular outcomes and mortality in sepsis.
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These features make C. batrachus a useful model for studying gene modulation responses, which may provide insights to better understand its adaptation mechanisms to survive under these adverse conditions.
Therefore, successful pharmacologic modulation of ERs may provide a potential target of future clinical studies of E2 for the treatment of CV and DCI following aneurysmal SAH.
In consideration of these two conflictive sepsis phases, modulation of Pellino3 may provide a new strategy for the development of a therapy approach in sepsis.
Conversely, modulation of hypoxia may provide benefit to patients with HNSCC [ 18], which underscores the importance of understanding the impact of therapies on tumor hypoxia and developing improved methods to modulate tumor pO2 and the molecular response to hypoxia.
Vibration-induced modulation of systemic hormones may provide a mechanism by which skeletal tissue is enhanced.
Thus, therapeutic modulation of preadipocyte differentiation may provide new insights that may lead to novel treatments for tissue regeneration and reconstructive medicine.
Taken together, these data suggest that specific modulation of SIRT1 activity may provide a therapeutic approach to pharmacologically control immune responses.
Modulation of their levels may provide a new approach for tackling myocardial necrosis.
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