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Its selective modulation is thought to alter the cancer-specific gene regulatory networks.
As such, response modulation is thought to influence one's situation selection (Gross & Thompson, 2007).
This modulation is thought to reflect changes in the weighting of information from each sensory modality, and should be considered in future electrophysiological investigations.
This modulation is thought to assist the ameloblasts in removing H+ ions from the enamel matrix, and its inhibition by F− is consistent with a decrease in the translation of proteins required for the modulation to occur.
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Factors influencing antigen and effector modulation are thought to be important in determining the final extent of depletion achieved (Table 1) [ 18, 27, 28].
Additionally, the results of the present study support Simons' integrated hypothesis of TrPs etiology, in which the autonomic modulation was thought to have a potential influence on the increase of ACh release, which can aggravate symptoms caused by TrPs [ 24].
The deficit in top-down modulation with age is thought to correspond to changes in the frontal and parietal lobes and the resulting decreased connectivity of the anterior-to-posterior network, in particular the frontal associative areas and the motor cortex [ 32, 33].
This experience-dependent modulation of the brainstem, which is thought to be under corticofugal control, occurs over the course of short-term (on the order of weeks) [20], [21] and lifelong auditory experience with behaviorally-relevant signals [1], [22], [23], [24], [25].
This combined with modulation of basal firing rates is thought to alter the signal-to-noise ratio of neuronal responses to synaptic input (Berridge and Waterhouse, 2003).
It is thought that this modulation of the state of the translation system results in the adjustment of nutrient consumption and to increased translation fidelity, two critical adaptations that apparently allow bacteria to survive starvation.
Modulation of cortical excitability via these pathways is thought to be important for the anticonvulsant effects of VNS [33].
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