Previous research has provided inconsistent results regarding the spatial modulation of auditory somatosensory interactions.
The model has provided examples of modifying loci (called modifiers of Min, Mom) in mice, demonstrating the principle of genetic modulation of disease severity represented by, in these cases, intestinal adenoma multiplicity.
Recent translational studies involving modulation of endothelial BMPR2 signaling have provided novel insights into treatment of PH, triggering a paradigm shift in our understanding of PH therapeutics (Fig. 1) (Long et al., 2015; Nickel et al., 2015; Prewitt et al., 2015).
The present results have provided evidence for modulation of the hepatic kynurenine pathway by disulfiram, possibly via its carbon disulphide metabolite.
These observations have provided a basis for estrogen modulation on KATP activity but the underlying mechanism is not fully understood.
Animal models have provided evidence for the importance of in vivo modulation of the immune system by the estrogen receptors [ 37, 38].
These findings have provided new insight into the molecular mechanism underlying the modulation of the KCNQ1/KCNE1 channel.
In conclusion, we have provided preliminary evidence of the immunomodulatory properties of iPSC-MSCs and BM-MSCs for the modulation of T-cell phenotypes in AR.
We have provided evidence that PD-L1 in antigen presenting DCs contributes to ligand-induced TCR down-modulation.
Lesion studies have provided evidence that the basal nuclear complex of the forebrain, the origin of cholinergic projections to the cerebral mantle, may be critical for the cholinergic modulation of learning and memory.
