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However, heterogeneity of extant findings suggests that it may be informative to elucidate individual difference factors that modulate the processing of emotional information.
Here, we investigated whether motivational incentives (monetary reward) modulate the processing of delayed intentions in the anterior prefrontal cortex (aPFC), known to be crucial for intention processing.
It is known that ADAR deaminase can modulate the processing and expression of microRNA and redirect the silencing targets (Blow et al., 2006; Kawahara et al., 2007b).
In the meantime, though, there have been many findings reported that are consistent with Dehaene et al.'s proposal and nowadays it is widely accepted in the masked priming literature that higher-level cognitive systems or resources (e.g. attention and expectation) can modulate the processing of subliminally presented stimuli [1], [8], [25], [28] [34].
While our results did not highlight any direct connection between the LC and the PFC, the former could indirectly modulate the processing of vigilance-related visceral cues in the PFC either through its effect on the amygdala or through striato-cortical loops [49].
We show that ADAR2 can modulate the processing of mir-376a2 independently of catalytic RNA editing activity.
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Heparan sulfate also can act indirectly in the system, e.g. by modulating the processing of growth factor precursors by matrix metalloproteases [25].
In contrast, the shutdown dissociation differentially modulates the processing of the unpleasant compared to neutral pictures.
One mechanism through which RARα signalling acts is by modulating the processing of APP into the non-amyloidic rather than the amyloidic pathway, without affecting the overall levels of APP.
Our results revealing the role of ROS in modulating the processing of pro-IL-1β (Fig. 7 and Supplementary Fig. S3) may partly explain the downstream effects of TLR2 on regulating joint inflammation shown in that study.
To evaluate the involvement of HSP70 in mediating the positive effects of BIIB021 on relieving proteasome impairment and possibly modulating the processing of PMP22, we used cells from a mouse model in which both inducible forms of HSP70, HSP70.1 and HSP70.3 (HSP70.1/3−/−), are deleted (Hunt et al., 2004).
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