Sentence examples for modulate paclitaxel sensitivity from inspiring English sources

We performed a loss-of-function RNAi screen to identify genes that modulate paclitaxel sensitivity [ 14].

These examples provide strong evidence that validate our identified druggable gene targets that modulate paclitaxel sensitivity.

The methods being evaluated were also applied to a real loss-of-function RNAi screening dataset to identify genes that modulate paclitaxel sensitivity in breast cancer cells.

We assessed promising methods using real data from a loss-of-function RNAi screen to identify hits that modulate paclitaxel sensitivity in breast cancer cells.

Based on the evaluation and comparison of the statistical approaches for analyzing high-throughput RNA interference (RNAi) screening simulation data, Ye et al., [ 8] assessed promising methods using real data from a loss-of-function RNAi screen to identify hits that modulate paclitaxel sensitivity in breast cancer cells.

To gain more mechanistic insight into the action of EB1 in modulating paclitaxel sensitivity, we transfected MCF7 cells with GFP or GFP-EB1 and then treated cells with paclitaxel (2 nmol/L).

Modulating miR-9 expression could change paclitaxel sensitivity of EOC cells.

Considering the potential of EB1 as a marker to predict paclitaxel sensitivity or as a target to increase paclitaxel sensitivity, the molecular mechanism of how EB1 regulates paclitaxel sensitivity merits further investigation.

We then sought to investigate the molecular mechanism of how EB1 regulates paclitaxel sensitivity.

In vitro cell proliferation assays reveal that EB1 stimulates paclitaxel sensitivity in breast cancer cell lines.

We then checked whether EB1 expression associates with paclitaxel sensitivity in breast cancer cell lines.