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The most plausible explanation in view of recent findings [ 7, 8] is that gut microbiota modulate mosquito response to Plasmodium.
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In Ae. aegypti, NPC1 may facilitate dengue virus entry while NPC2 proteins may play a role in modulating mosquito immunity to dengue virus [ 91].
The oscillating pattern of oxidative phosphorylation gene expression may modulate the mosquito's ability to withstand insecticide exposure.
The oscillating pattern of oxidative phosphorylation gene expression may modulate the mosquito's ability to withstand insecticide exposure and suggests the interesting possibility that susceptibility to insecticides may show diurnal fluctuations.
High spatial and temporal variability has been reported in field-derived feeding indices, and this may be expected because interactions with complex biotic and abiotic factors in nature modulate the mosquitoes' innate host preferences.
In addition, we can modulate the daily mosquito-emergence rate as a function of the size of the adult population.
Recent laboratory studies showed that female and male mosquitoes modulate their flight tone harmonics to match that of their mate [ 40- 42].
It suggested that a part of viral RNAs may be degraded at the early phase of infection, likely to modulate virus growth, in mosquito cells.
When considering these pathways, it is important to keep in mind that evolutionary pressure exists on both the arbovirus and the vector mosquito to modulate the immune response.
Microfilarial proteins likely to modulate the vertebrate and mosquito immune response are those that are likely to be secreted by or on the surface of the microfilariae.
The possibility that pre-exposure to mosquito saliva might modulate WNV infection was explored.
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