This is consistent with the model that chromatin plasticity is a key feature for genome expression [2], [44].
Chromatin architectural proteins interact with nucleosomes to modulate chromatin accessibility and higher-order chromatin structure.
The DCSR pairs represent the locations of imbalances of chromatin accessibility between alleles and are ideal to reveal chromatin motifs that may directly modulate chromatin accessibility.
Results obtained in this study may also be relevant for our understanding of chromatin plasticity during metazoan embryogenesis.
How do chromatin plasticity and the underlying enzymatic machinery contribute to memory formation in health and disease?
The ability to describe these varying degrees of contribution is essential for an accurate understanding of chromatin plasticity.
Like smaller RNAs, large ncRNAs (> 1 kb) can also modulate chromatin states and gene expression.
Therefore, Elongator may also modulate chromatin at defense loci in Arabidopsis through DNA methylation.
Cells utilize two distinct mechanisms to modulate chromatin dynamics during DNA repair.
HOTAIR regulates gene expression by modulating chromatin structures.
