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By explicitly considering the consequences for infection instability, we were able to deduce analytically those critical penetrance levels above which the spread of a modifier leads to loss of Wolbachia.
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During the synthesis of these frameworks, the addition of 4,4′-bipyridine as a topological modifier led to the formation of the pto polymorph, which saw 40% less nitrogen uptake than the tbo polymorph, due to a decrease in accessible surface area.
The presence of PMSF (a serine modifier) and PAO (a cysteine modifier) led to the inactivation of the enzyme, which meant that serine and cysteine residues were essential for its catalytic function.
The involvement of modifiers leads to the discussion of the variation in the forms of minimizers.
A comparison of the results for different modifiers leads to the conclusion that steric repulsion caused by the anchoring group plays an important role in the enantiodifferentiating interaction.
Knockout experiments show that the deletion of DNA methyltransferase or histone modifiers leads to embryonic lethality.
After reductive pretreatment the modifiers suppress the hydrogen consumption, while after combined reductive pretreatment the modifiers lead to the promotion of both oxidation reactions.
The rise of the reactivity R of the organic component of OIS by increasing the content of the isocyanate-containing modifier PIC leads to the formation of more rigid, thermostable, less conductive and polarisable OIS.
Therefore, it is likely a complex interaction between mutation type, XCI skewing (in the case of females), and other genetic modifiers that leads to the final phenotype in MECP2 mutations.
It was found that longer PDMS segments in PER PDMS PER and higher modifier contents led to lower μs of modified epoxy resins upon stainless steel.
Generation of cytokines, chemokines, and ECM modifiers by senescent cells leads to death of cells around them, tissue remodeling, and attraction of immune elements.
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CEO of Professional Science Editing for Scientists @ prosciediting.com