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Therefore, current work in our lab is investigating how GLP-1 receptor signaling improves islet function after bariatric surgery using various genetically modified mouse models.
An important advance in the study of Huntington's disease has been the development of genetically modified mouse models.
Numerous genetically modified mouse models have recently been developed for the study of the pituitary gonadal interactions.
Therefore, we have ongoing studies in various genetically and pharmaceutically modified mouse models investigating how TGR5 contributes to the effect of bariatric surgery to improve these obesity co-morbidities.
For this purpose, the use of OSCC mouse models, such as chemically induced mouse models, genetically modified mouse models, and transplanted (xenograft) models, is crucial.
Inbred strains of mice have served as valuable models for studying genetic susceptibility to drug addiction, an alternative to genetically modified mouse models.
The advent of mammalian gene engineering and genetically modified mouse models has led to renewed interest in developing resources for referencing and quantitative analysis of mouse brain anatomy.
With the advent of genetically modified mouse models of disease, validated tests provide an opportunity to screen mouse models of disease for early onset cognitive deficits.
Genetically modified mouse models have demonstrated the importance of miRNA pathways for normal spermatogenesis, and functional studies have been designed to dissect the roles of specific miRNAs in distinct cell types.
This review will summarize the major findings obtained with these two genetically modified mouse models and briefly discuss the similarities and differences between them and with the human phenotype.
Use of genetically modified mouse models for evaluation of carcinogenic risk: considerations for the laboratory animal scientist.
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