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Strong predictors for the risk of cardiovascular disease in older adults include levels of total cholesterol, low-density lipoprotein cholesterol, and high-density lipoprotein cholesterol, which can be modified by dietary behavioral changes and regular physical exercise.
Given the close association between low education and unhealthy eating habits (i.e. a high GI diet), it might be possible that the influence of SNP rs3809508 on obesity risk was, at least partly, modified by dietary GI.
Thus, Hcy status in plasma can be modified by dietary interventions.
Furthermore, this association appeared to be modified by dietary intake, particularly intake of isoflavone.
This association appeared to be modified by dietary intake, particularly isoflavone.
In the case of mitROSp, it is modified by dietary manipulations that increase mean and maximum longevity.
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AUF1 isoforms p40 and p42 are modified by dietary-induced hypocalcemia, and the secondary hyperparathyroidism of chronic kidney disease and its treatment by R568 [ 7, 27].
In contrast, plasma levels of triglycerides and free fatty acids were not significantly modified by the dietary treatments (Table 1).
However, we are not aware of any study which has examined whether associations between SNPs of hypothalamic genes and weight gain are modified by these dietary factors.
For example, susceptibility to lead toxicity can be modified by several dietary factors.
Both telomere length and hypertension risk can be modified by certain dietary factors, such as fruit and vegetables.
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