Sentence examples for modifications in young from inspiring English sources

Since environmental modifications in young animals may result in permanent alterations in neuroendocrine function, the present study was designed to determine the effect of a space flight on oxytocinergic and vasopressinergic magnocellular hypothalamic neurons of prepuberal rats.

Table S3 Posttranslational modifications in young mice.

Nonetheless, it has been suggested that aggressive risk factor modification in young people is justifiable and may in fact prevent, as opposed to merely decreasing the risk of, clinically overt cardiovascular disease.

The significant weight gain after 20 years of age in patients with abdominal obesity stresses the importance of lifestyle modification in younger generation, to prevent potential development of type 2 diabetes and future atherosclerotic cardiovascular disease.

19– 24 Other than a large audit of German patients with type 1 diabetes, 22 there is little data on the achievement of treatment targets and cardiovascular risk factor modification in younger patients with diabetes.

These results suggest that education on lifestyle modification in younger generation, to prevent visceral fat accumulation is important for the prevention of type 2 diabetes and future atherosclerotic cardiovascular disease.

The relative abundance of oxidative modifications (carbonyls) in young, middle-aged and aged CKm preparations were determined using the Oxyblot kit (Intergen Company; 18, 19).

The most relevant finding is the identification of profile modifications detected in young 3xTg-AD mice that closely resemble those occurring upon physiological aging in WT animals, thereby suggesting that 3 m.o.o

A spectrum of developmental and behavioral modifications in adolescent, young-adult and aged animals resulted after prenatal nicotine exposure.

This is consistent with our observation that the declining levels of oxidative modification in the young correlate with the normalization of GAPDH pool levels whereas for the old animals normalization of GAPDH pool levels is significantly delayed, i.e., were not reached by 7 days in spite of evidence for increased GAPDH transcription.

Consistent with the findings in frataxin-overexpressing cells, we could inversely show that the steady-state level of Fpg-sensitive DNA modifications in hepatocytes isolated from young frataxin-deficient AlbFxn−/− mice was increased, notably in the absence of any external ROS-inducing or mutagenic stimulus.