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These modifications in turn induce an increase in the strong ion difference and in the plasma bicarbonate concentration [ 14].
Consider these modifications in turn.
These modifications in turn may translate into enhanced collagen phagocytosis and reorganization, processes that are essential for connective tissue homeostasis.
These site-specific modifications, in turn, will be associated with changes in the expression of different genes with diverse effects.
These modifications in turn recruit the SET3C/RPD3S histone deacetylase complexes to create a chromatin environment repressive for transcription initiation [ 61- 63].
These possible modifications, in turn, are likely to lead to the wrong far from the true model (Asparouhov and Muthén 2014).
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This modification, in turn, leads to a change in membrane permeability, as observed by surface enhanced Raman scattering.
Altered histone modification, in turn, is associated with altered gene expression (Pal-Bhadra et al. 2007).
We propose a model whereby the interaction of Nup98-HoxA9 with prebound Crm1 induces the alteration of chromatin structures specifically on Hox cluster regions and this modification, in turn, causes robust activation of gene expression.
EZH2 acts as a lysine-specific HMT that mediates H3K27me3; this modification, in turn, provides a specific binding site for another PRC, PRC1, to silence the expression of PRC2 target genes by blocking the recruitment of transcriptional activation factors and preventing initiation of transcription (4, 5).
It is possible that lack of NCAM results in agitation of neuron glia interactions, and modifications in these interactions might in turn be responsible for the inhibition of migration in the RMS.
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