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Given that the mutants were engineered to abolish key protein intra-molecular interaction, it is highly probable that the modifications could lead to changes in the structural stability of these GST-C variants.

However, such modifications could lead to the amplification of non-orthologous closely related loci.

On the other hand, some genetic modifications could lead to more intensive livestock production with associated increases in environmental pollutants.

These modifications could lead to enhancements and an automated sampling analysis system coupled to MS, using aqueous media.

The detection of insoluble SOD1 in ALS patients suggests that mutations and modifications could lead to conformational changes in SOD1 that, in turn, increase the chances that it will misfold and form insoluble aggregates (Bosco et al., 2010).

Further modifications could lead to improvements for C. bescii as both a microorganism for consolidated bioprocessing and as an expression host for secretion of heterologous proteins especially those originating from thermophiles.

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As deduced from the Rosensweig equation and under certain experimental conditions, both Néel and Brownian relaxation times are comparable for SPM nanoparticles around 10 nm; therefore, changes in the particle concentration, solvent viscosity or particle surface modification could lead to important differences in the SAR observed.

This protocol modification could lead to better discrimination of signal intensity drops across regions with an indel.

Such effect modification could lead to nondifferential misclassification bias (trials are conducted in random order), resulting in an underestimation of the true effect that pollutant exposure has on the induction of inflammation in humans.

Usually, the modelling of micro-machining by pulsed laser ablation is done using a pulse-by-pulse evaluation of the surface modification, which could lead to inaccuracies when pulses overlap.

Alterations of the chromatin modification proteins could lead to disruptions of transcriptional regulation and tumour formation [ 92].

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