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The reduction of Ce(IV) to Ce(III) and surface modification seem to be responsible for de-activation of the oxide.
Changes in histone modification seem to be closely involved in the complex changes in gene expression that drive early development [10].
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The peptide surface modification seems to be biocompatible.
The key to receiving a modification seems to be convincing the bank that you're in its modification "sweet spot".
Rather, this particular histone modification seems to be associated with the proliferative, undifferentiated cellular state of the neural precursor cells.
Several pumpkin phloem exudate proteins appear to be post-translationally modified by phosphorylation and/or glycosylation and their modification seems to be required for the interaction of non-cell-autonomous phloem proteins with chaperones, enabling their movement through plasmodesmata [ 46].
Although air quality forecasting efforts have so far been focused on concentration predictions, forecasting health benefit sensitivities for guiding short-term emission modification seems to be the next logical step.
So far, the modifications seem to be working.
Age-associated modifications seem to be regulated in a cell type specific manner.
In addition to direct control of gene transcription, post-transcriptional modifications seem to be very important for T cell development, homeostasis and activation as well.
These two post-translational modifications seem to be linked since, in at least some cases, tyrosine phosphorylation is monoubiquitination-dependent [11].
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com