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A highly valued cooking and baking fat, lard is blended, frequently after modification by molecular rearrangement or hydrogenation, with other fats and oils to make shortening.
Its recent, lengthy report on the matter can be summarized in one sentence: Crop modification by molecular methods is no more dangerous than crop modification by other methods.
To our knowledge, only one previous study on smoking and CRC survival has considered potential effect modification by molecular phenotypes of tumour (Phipps et al, 2011).
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High-resolution DBD structures comprising the intact wing motif determined in our study served as good starting models for studying the structural effects of these important modifications by molecular dynamics (MD) simulation.
Our results suggest that comprehensive analysis of the relationship between the degree of modification by polymer, molecular size, and specific activity could lead to the development of bioconjugated proteins with increased therapeutic value and decreased side effects.
A pattern seems to appear in the taxonomic modifications revealed by molecular phylogenies in Ficus, suggesting that they do not result from identification/manipulation errors or from limiting phylogenetic information.
In this Perspective, we focus on three types of interfacial modification: band alignment by molecular dipole layers, improved CQD film mobilities by ligand exchange, and reduced recombination by interfacial inorganic layers.
Since the prediction based on classical NIEL model cannot fit low energy incident well, low energy particles induced displacement damage mechanism, defect generation, recombination and effective NIEL modification is discussed by molecular dynamics (MD) model.
In this context, the proteins and domains that recognize histone modifications, named "effectors" or "readers", are thought to define the functional consequences of many classes of modifications by transducing molecular events at chromatin into biological outcomes.
A bioprecursor prodrug is a compound that is metabolized by molecular modification into a new compound, which is the active principle to the active drug.
This is the first report of a gene therapy vector engineered to discriminate between infection in malignant and normal cells by molecular modification of a tumor-specific protein.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com