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Recently, O-GlcNAc stimulation (a post-translational modification) appeared to be beneficial in acute pathologies.
This modification appeared to bias Lats2 kinase activity toward inhibitory phosphorylation of Yap at serine 381 but not serine 127 [57].
Post-translational modification appeared to be an important element of HIV-1 cellular network hijacking.
Based on these results, stratified analyses were undertaken on variables for which effect modification appeared to be present.
The degree of behaviour modification appeared to vary according to the severity of the harm that participants anticipated occurring as a result.
The mmLDL modification appeared to be very reproducible, and a successful modification was documented in a biological assay in which mmLDL induced spreading of J774 macrophages in cell culture [ 13, 19].
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A strong outgrowth of axons was found, and such a surface modification appears to be suitable for the creation of neuronal patterns with effective growth of axons in vitro.
Although transcriptional repression by histone modification appears to contribute to the process of cellular differentiation, it is unclear what mediates the specificity of PcG proteins.
Because the high-temperature component displays linear characteristics during progressive thermal demagnetization (Fig. 3), the modification appears to have occurred once at around 450°C during the cooling.
The motor for this modification appears to be the approach of the GEF.
Thus the Oto modification appears to be independent of C77 palmitoylation.
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