Given the suggestion that interventions aimed at modifying early weight gain could prevent adult obesity [ 1], our aim was to determine which modifiable risk factors, especially those related to feeding practices or behaviours, are associated with rapid weight gain in early infancy.
Modifiable risk factors are weight gain, insulin resistance, and diabetes, and nonmodifiable are age, race, genetic background, and baseline histology.
Consequently, prevention of unnecessary weight gain should target modifiable behaviours that influence energy intake and expenditure including diet, PA and SB.
Like the peripheral pO2 receptor, the central pCO2 receptor set point and gain are sympathetically modifiable [ 58]; suggesting that the clinical appreciation of dyspnea may reflect sympathetic hyperactivity quite independent of the mechanism by which alveolar and interstitial lung water stimulate interstitial pulmonary juxtacapillary receptors.
This study will provide evidence on the potential health gains of a modifiable cardiovascular risk factor screening program for women whose pregnancy was complicated by hypertension or preeclampsia.
Therefore, specific recommendations should be created since gestational weight gain could be a modifiable risk factor for adverse obstetric outcomes.
As obesity has reached epidemic proportions and is also a modifiable lifestyle factor, avoidance of weight gain throughout life is a salient measure for prevention of breast cancer.
In this analysis the two main modifiable factors associated with rapid weight gain in early infancy were formula feeding and feeding on schedule.
Prevention of unnecessary weight gain should thus target modifiable energy intake and energy expenditure behaviours, i.e. physical activity, sedentary, and dietary behaviours, also referred to as energy balance related behaviours (EBRBs).
Male gender and lower birthweight were non-modifiable factors associated with rapid weight gain.
We sought to identify modifiable risk factors for excessive gestational weight gain (GWG).
