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Failure loads, load mid-span defload mid-spanionships andeflectionmodes of cellularelationships were predicted from the finite element andlysis.
The present study reports on the results of a series of computational experiments that explore the effect of topology and microstructural irregularity (or non-periodicity) on deformation modes of cellular structures under both uniaxial and biaxial stress states.
Whilst there are many modes of cellular regulation, membrane bound receptors represent an attractive target for small molecule-mediated control of cellular activity because of their accessibility.
The other three sub-models describe mechanisms that are generally applicable to all modes of cellular senescence.
The parameter ACTIVATION THRESHOLD reflects a threshold dividing the two distinct modes of cellular behavior, in stay and in move.
Summarising, the responses discussed here all represent plausible modes of cellular disruption, and all of them have been demonstrated previously to be induced by copper exposure.
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Extremely long or strong stress may induce a topological phase transition in the respective cellular networks, which switches the cell to a completely different mode of cellular function.
Using flow cytometry and confocal microscopy, we have examined the internalisation of Sj.GST26 into live cells under a variety of conditions designed to shed light on the mode of cellular uptake.
The HSP81.3 and 81.1, of the HSP90 gene family, are associated with different polypeptides serving a general mode of cellular activities.
Non-fusogenity with lysosomes is controlled by the mode of cellular uptake [6], [7] and chlamydial protein factors [8], [9].
The adaptive stress response of bacteria is a crucial mode of cellular protection and allows bacteria to survive in changing environments.
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