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In conclusion, comparing gene expression levels in moderate specimens, including normal liver tissues, paired tumor and non-tumor tissues of patients with HCC (including different status of metastasis), our research revealed two appropriate genes TBP and HPRT1 were reliable for normalization in the study of HBV-related HCC.
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39 In a previous study to identify molecular markers of prognosis in chronic hepatitis C, mRNA expression was quantified by real-time quantitative RT-PCR in a large number of selected genes in F2 (moderate fibrosis) specimens and compared with F1 (mild fibrosis) specimens.
For the inducible HSP70, a few of the mild and moderate activity epithelial specimens showed weak staining on the surface; the severe activity specimens showed almost no staining.
In FCD specimens, moderate to strong VEGFR-1 staining was observed within the dysplastic cortex (Fig. 4c g; Table 2; Fig. 6).
In FCD specimens, moderate to strong VEGFB IR was observed within the dysplastic cortex (Fig. 3b; Table 2; Fig. 6) with strong VEGFB IR in dysplastic neurons (Fig. 3c).
Cellularity of the direct smears with the new needle was evaluated semi-quantitatively (hypocellular- very few scattered cells, mild- <20% area in the field covered by cells in the specimen, moderate- 20%to50%0% area in the field covered by cells in the specimen, and marked- >50% area in the field covered by cells in the specimen).
All 2+ (moderate-intensity) staining specimens underwent FISH analysis and those who showed HER2 amplification were also classified as positive by the methods described earlier (Bartlett et al, 2003).
The EIFS values agreed well with the median corrosion-pit depths measured on the exposed specimens with moderate corrosion.
The results show that the bond stiffness and strength tend to increase for the specimens exhibiting moderate ASR deterioration, but decrease as ASR deterioration reaches a certain level.
These specimens represented moderate to severe OA.
These specimens represented moderate to severe OA [ 34].
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