For higher credibility and accuracy, quantum-mechanical models are activated and empirical material parameters are employed from the existing literature.
The oncogenic K-ras alleles in these models are activated somatically in one (KrasLA1) and conditionally in the other (KrasLSL) (5, 7).
The neurons in the hidden layer in these models are activated not only by the input neurons but also by the neighboring neurons in the layer; consequently, the dynamics of these neuron activities are not determined solely by the inputs but can autonomously change even in the absence of inputs, as in the case of our model.
Therefore, Re θc is the point where the model is activated to match both Re θt and F length.
Instead, an elastic degrading material model is activated for the failed layer to simulate the post-ultimate response.
The contact model is activated during the contact phase which is defined by both geometrical and force-dependent conditions.
The final output of the module is the level of caspase-3, which (in the model) is activated by Bax (or Bak), but also auto-catalytically, which renders the apoptotic decision irreversible.
In our in vitro model system, PMNs were activated, as evidenced by the production of hROS [ 9], inhibited HSV spread, and did not support HSV growth.
The parametric model did not update the orthosis model after the toggles were activated, e.g., the thickening or engraving function failed, or sometimes it generated a valid model that had a distorted surface or separate objects on the shell.
In subtype classification model, the two classes were activated B-cell-like subtype and germinal center B-cell-like subtype.
