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Using merlin-mutant mouse models, we therefore investigated whether any Schwann cell changes could be detected in vivo.

Estimates of β were very similar between age-adjusted and multivariate-adjusted models; we therefore present the multivariate-adjusted results only.

In view of the predicted highly significant temperature increases by all existing climate models, we therefore postulated that these increases will coincide with extended annual time spans for tularaemia outbreaks.

In fitting explicitly additive and multiplicative models we therefore checked for predictions beyond the feasible range and implemented in the Stata procedure gllamm (Rabe-Hesketh, Skrondal & Pickles, 2004), an iterative estimation method in which the residual variance was made a linear function of the predicted proportion.

Because deleterious mutations are known to circulate in the influenza A/H3N2 virus population (Fitch et al., 1997; Pybus et al., 2007; Strelkowa and Lässig, 2012), and in light of existing criticisms of the other two models, we therefore argue that the model presented here provides a more plausible mechanistic explanation for influenza's characteristic evolutionary features.

Similar(55)

On the basis of the findings from the measurement model, we therefore constructed scales by averaging the item scores.

It sequentially reconstructs a signal sequence following our linear dynamic model, we therefore use it to implement the image reconstruction process of our LDSM method.

For the explanatory model, we therefore only consider 718 objects for which a realized market price is known, here from foreclosure auctions and over-the-counter trades.

In the regression model, we therefore control for a range of variables, namely, demographic characteristics, institutional education level, and household income, experience of illness among household members, occupation and place of residence.

To simplify the model, we therefore removed this part of the plants genome.

Based on this model, we therefore hypothesised that upregulation of CEP55 and HELLS precedes gene amplification during malignant transformation induced by FOXM1B.

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